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intraperitoneal telemetric ecg transmitter  (Data Sciences International)

 
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    Data Sciences International intraperitoneal telemetric ecg transmitter
    Intraperitoneal Telemetric Ecg Transmitter, supplied by Data Sciences International, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/telemetric+transmitters+intraperitoneally/intraperitoneal+telemetric+ecg+transmitter/pm38849501-244-6-9
    Average 90 stars, based on 1 article reviews
    intraperitoneal telemetric ecg transmitter - by Bioz Stars, 2026-10
    90/100 stars

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    Activity Assay:

    Article Title: Glutamatergic Signaling from the Parabrachial Nucleus Plays a Critical Role in Hypercapnic Arousal
    Article Snippet: .. These mice were also then instrumented with EEG and EMG electrodes and were implanted with telemetric transmitters intraperitoneally for the recording of EKG, body temperature and locomotor activity (PhysioTel® ETA-F10, Data Science International, MN). ..



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    a Survival curve of CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T (43 vs. 64 animals, median survival 72 vs. 98 days, blinded analysis). Log-rank (Mantel–Cox test and Gehan–Breslow–Wilcoxon test (two-tailed analysis) were performed to calculate the survival percentage of mice. Probability vs CaMKIIδc +/T . b Hearts from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. c Ratio of heart weight to tibia length as a parameter of cardiac hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in this ratio compared to WT and SCN10A −/− mice. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. ( N = hearts studied, WT = 14, SCN10A −/− = 16, CaMKIIδc +/T = 13, and SCN10A −/− /CaMKIIδc +/T = 23). Data were presented as mean values ± SEM. d Original histological wheat germ agglutinin staining from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. Scale bars = 75 µm. Stainings were produced from different sections and three different regions (basal, mid-ventricular, and apical) of each heart studied. e Cardiomyocyte cross-sectional-area (CSA) as a parameter for cellular hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in CSA compared to WT and SCN10A −/− mice. CSA in CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T mice did not significantly differ. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. N = hearts studied (>300 cardiomyocytes were studied per heart, from different sections and different regions (basal, mid-ventricular, apical), WT = 5 hearts, SCN10A −/− = 5 hearts, CaMKIIδc +/T = 4 hearts, SCN10A −/− /CaMKIIδc +/T = 5 hearts. Data were presented as mean values ± SEM. f Original <t>echocardiography</t> recordings from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T at M-mode in 12–13- week-old mice. g Echocardiography recordings revealed a decrease in left ventricular ejection fraction (EF) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001(one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM. h Echocardiography recordings revealed a significant increase in left ventricular end-diastolic diameter (LVEDD) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001). LVEDD was not significantly different in WT vs SCN10A −/− or CaMKIIδc +/T vs SCN10A −/− /CaMKIIδc +/T (one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM.
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    a Survival curve of CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T (43 vs. 64 animals, median survival 72 vs. 98 days, blinded analysis). Log-rank (Mantel–Cox test and Gehan–Breslow–Wilcoxon test (two-tailed analysis) were performed to calculate the survival percentage of mice. Probability vs CaMKIIδc +/T . b Hearts from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. c Ratio of heart weight to tibia length as a parameter of cardiac hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in this ratio compared to WT and SCN10A −/− mice. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. ( N = hearts studied, WT = 14, SCN10A −/− = 16, CaMKIIδc +/T = 13, and SCN10A −/− /CaMKIIδc +/T = 23). Data were presented as mean values ± SEM. d Original histological wheat germ agglutinin staining from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. Scale bars = 75 µm. Stainings were produced from different sections and three different regions (basal, mid-ventricular, and apical) of each heart studied. e Cardiomyocyte cross-sectional-area (CSA) as a parameter for cellular hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in CSA compared to WT and SCN10A −/− mice. CSA in CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T mice did not significantly differ. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. N = hearts studied (>300 cardiomyocytes were studied per heart, from different sections and different regions (basal, mid-ventricular, apical), WT = 5 hearts, SCN10A −/− = 5 hearts, CaMKIIδc +/T = 4 hearts, SCN10A −/− /CaMKIIδc +/T = 5 hearts. Data were presented as mean values ± SEM. f Original <t>echocardiography</t> recordings from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T at M-mode in 12–13- week-old mice. g Echocardiography recordings revealed a decrease in left ventricular ejection fraction (EF) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001(one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM. h Echocardiography recordings revealed a significant increase in left ventricular end-diastolic diameter (LVEDD) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001). LVEDD was not significantly different in WT vs SCN10A −/− or CaMKIIδc +/T vs SCN10A −/− /CaMKIIδc +/T (one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM.
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    a Survival curve of CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T (43 vs. 64 animals, median survival 72 vs. 98 days, blinded analysis). Log-rank (Mantel–Cox test and Gehan–Breslow–Wilcoxon test (two-tailed analysis) were performed to calculate the survival percentage of mice. Probability vs CaMKIIδc +/T . b Hearts from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. c Ratio of heart weight to tibia length as a parameter of cardiac hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in this ratio compared to WT and SCN10A −/− mice. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. ( N = hearts studied, WT = 14, SCN10A −/− = 16, CaMKIIδc +/T = 13, and SCN10A −/− /CaMKIIδc +/T = 23). Data were presented as mean values ± SEM. d Original histological wheat germ agglutinin staining from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. Scale bars = 75 µm. Stainings were produced from different sections and three different regions (basal, mid-ventricular, and apical) of each heart studied. e Cardiomyocyte cross-sectional-area (CSA) as a parameter for cellular hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in CSA compared to WT and SCN10A −/− mice. CSA in CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T mice did not significantly differ. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. N = hearts studied (>300 cardiomyocytes were studied per heart, from different sections and different regions (basal, mid-ventricular, apical), WT = 5 hearts, SCN10A −/− = 5 hearts, CaMKIIδc +/T = 4 hearts, SCN10A −/− /CaMKIIδc +/T = 5 hearts. Data were presented as mean values ± SEM. f Original echocardiography recordings from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T at M-mode in 12–13- week-old mice. g Echocardiography recordings revealed a decrease in left ventricular ejection fraction (EF) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001(one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM. h Echocardiography recordings revealed a significant increase in left ventricular end-diastolic diameter (LVEDD) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001). LVEDD was not significantly different in WT vs SCN10A −/− or CaMKIIδc +/T vs SCN10A −/− /CaMKIIδc +/T (one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM.

    Journal: Nature Communications

    Article Title: Detrimental proarrhythmogenic interaction of Ca 2+ /calmodulin-dependent protein kinase II and Na V 1.8 in heart failure

    doi: 10.1038/s41467-021-26690-1

    Figure Lengend Snippet: a Survival curve of CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T (43 vs. 64 animals, median survival 72 vs. 98 days, blinded analysis). Log-rank (Mantel–Cox test and Gehan–Breslow–Wilcoxon test (two-tailed analysis) were performed to calculate the survival percentage of mice. Probability vs CaMKIIδc +/T . b Hearts from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. c Ratio of heart weight to tibia length as a parameter of cardiac hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in this ratio compared to WT and SCN10A −/− mice. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. ( N = hearts studied, WT = 14, SCN10A −/− = 16, CaMKIIδc +/T = 13, and SCN10A −/− /CaMKIIδc +/T = 23). Data were presented as mean values ± SEM. d Original histological wheat germ agglutinin staining from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T mice. Scale bars = 75 µm. Stainings were produced from different sections and three different regions (basal, mid-ventricular, and apical) of each heart studied. e Cardiomyocyte cross-sectional-area (CSA) as a parameter for cellular hypertrophy. CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T showed a significant increase in CSA compared to WT and SCN10A −/− mice. CSA in CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T mice did not significantly differ. Data were analyzed by one-way ANOVA with post hoc Bonferroni’s correction. N = hearts studied (>300 cardiomyocytes were studied per heart, from different sections and different regions (basal, mid-ventricular, apical), WT = 5 hearts, SCN10A −/− = 5 hearts, CaMKIIδc +/T = 4 hearts, SCN10A −/− /CaMKIIδc +/T = 5 hearts. Data were presented as mean values ± SEM. f Original echocardiography recordings from WT, SCN10A −/− , CaMKIIδc +/T , and SCN10A −/− /CaMKIIδc +/T at M-mode in 12–13- week-old mice. g Echocardiography recordings revealed a decrease in left ventricular ejection fraction (EF) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001(one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM. h Echocardiography recordings revealed a significant increase in left ventricular end-diastolic diameter (LVEDD) in CaMKIIδc +/T (six mice) and SCN10A −/− /CaMKIIδc +/T (six mice) compared to WT (seven mice) or SCN10A −/− (eight mice) ( p < 0.0001). LVEDD was not significantly different in WT vs SCN10A −/− or CaMKIIδc +/T vs SCN10A −/− /CaMKIIδc +/T (one-way ANOVA with post hoc Bonferroni’s correction). Data were presented as mean values ± SEM.

    Article Snippet: Mice were implanted with an intraperitoneal telemetric ECG transmitter (TA11ETA-F10, Data Sciences International) with its Cable in lead II configuration.

    Techniques: Two Tailed Test, Staining, Produced

    a Original ECG traces from telemetry recordings of 10-week-old CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T mice showing ventricular arrhythmias. b Unchanged activity levels in SCN10A −/− /CaMKIIδc +/T compared to CaMKIIδc +/T (CaMKIIδc +/T (four mice); SCN10A −/− /CaMKIIδc +/T (three mice), four individual recordings each). Data were presented as mean values ± SEM, analyzed by unpaired two-tailed Student’s t -test. c Mean values of premature ventricular contractions (PVCs) in SCN10A −/− /CaMKIIδc +/T ( p = 0.08, Unpaired two-tailed Student’s t -test), CaMKIIδc +/T (four mice); SCN10A −/− /CaMKIIδc +/T (three mice), four individual recordings each. Data were presented as mean values ± SEM. d Reduction of ventricular tachycardia (VT) incidence in SCN10A −/− /CaMKIIδc +/T ( p < 0.05, Unpaired two-tailed Student’s t -test), CaMKIIδc +/T (four mice); SCN10A −/− /CaMKIIδc +/T (three mice), four individual recordings each. Data were presented as mean values ± SEM.

    Journal: Nature Communications

    Article Title: Detrimental proarrhythmogenic interaction of Ca 2+ /calmodulin-dependent protein kinase II and Na V 1.8 in heart failure

    doi: 10.1038/s41467-021-26690-1

    Figure Lengend Snippet: a Original ECG traces from telemetry recordings of 10-week-old CaMKIIδc +/T and SCN10A −/− /CaMKIIδc +/T mice showing ventricular arrhythmias. b Unchanged activity levels in SCN10A −/− /CaMKIIδc +/T compared to CaMKIIδc +/T (CaMKIIδc +/T (four mice); SCN10A −/− /CaMKIIδc +/T (three mice), four individual recordings each). Data were presented as mean values ± SEM, analyzed by unpaired two-tailed Student’s t -test. c Mean values of premature ventricular contractions (PVCs) in SCN10A −/− /CaMKIIδc +/T ( p = 0.08, Unpaired two-tailed Student’s t -test), CaMKIIδc +/T (four mice); SCN10A −/− /CaMKIIδc +/T (three mice), four individual recordings each. Data were presented as mean values ± SEM. d Reduction of ventricular tachycardia (VT) incidence in SCN10A −/− /CaMKIIδc +/T ( p < 0.05, Unpaired two-tailed Student’s t -test), CaMKIIδc +/T (four mice); SCN10A −/− /CaMKIIδc +/T (three mice), four individual recordings each. Data were presented as mean values ± SEM.

    Article Snippet: Mice were implanted with an intraperitoneal telemetric ECG transmitter (TA11ETA-F10, Data Sciences International) with its Cable in lead II configuration.

    Techniques: Activity Assay, Two Tailed Test